Tumor necrosis factor, cytokines, and the hyperlipidemia of infection.

Tumor necrosis factor, cytokines, and the hyperlipidemia of infection. Research Abstract Details 

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Tumor necrosis factor, cytokines, and the hyperlipidemia of infection. Abstract Text:

The hypertriglyceridemia of infection is produced by many of the cytokines that regulate the immune response, including the tumor necrosis factors, interleukin-1, interleukin-6, and the interferons. In rodents, this hypertriglyceridemia is primarily due to the ability of these cytokines to stimulate hepatic lipogenesis and very low density lipoprotein production without causing significant changes in adipose tissue lipoprotein lipase or triglyceride clearance. Induction of hypertriglyceridemia is not directly linked to the syndrome of wasting. Rather, the increase in hepatic production of lipoproteins is part of the acute-phase response and may contribute to host defense.

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MEDLINE DATE: 1991 Nov-Dec

Tumor necrosis factor, cytokines, and the hyperlipidemia of infection. Journal Published:

PUBLICATION TYPE: Journal Article

Journal: Trends in endocrinology and metabolism: TEM

VOLUME: 2

Page Numbers: 213-9

Journal Abbreviation: Trends Endocrinol. Metab.

ISSN: 1043-2760

DAY: 15

MONTH: 04

YEAR: 2008

Tumor necrosis factor, cytokines, and the hyperlipidemia of infection. Information

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LANGUAGE: eng

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Grant and Affiliation Information for Tumor necrosis factor, cytokines, and the hyperlipidemia of infection.

AFFILIATION: Carl Grunfeld and Kenneth R Feingold are at the Department of Medicine, University of California, San Francisco, Metabolism Section, Medical Service, USA; Department of Veterans Affairs Medical Center, San Francisco, CA 94121, USA.

Country: United States

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MEDLINETA: Trends Endocrinol Metab

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