The gene balance hypothesis: from classical genetics to modern genomics.

The gene balance hypothesis: from classical genetics to modern genomics. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

The gene balance hypothesis: from classical genetics to modern genomics. Abstract Text:

James A Birchler,Reiner A Veitia,

The gene balance hypothesis: from classical genetics to modern genomics. Publishing Authors By Initials

JA Birchler,RA Veitia,

For similar genetic phenomena: ploidies research abstracts see: genetic phenomena: ploidies research

PUBMED ID PMID:

MEDLINE DATE:

The gene balance hypothesis: from classical genetics to modern genomics. Journal Published:

PUBLICATION TYPE: Review

Journal: The Plant cell

VOLUME: 19

Page Numbers: 395-402

Journal Abbreviation: Plant Cell

ISSN: 1040-4651

DAY: 9

MONTH: 02

YEAR: 2007

The gene balance hypothesis: from classical genetics to modern genomics. Information

Number of References: 70

LANGUAGE: eng

NlmUniqueID: 9208688

The gene balance hypothesis: from classical genetics to modern genomics. Keywords Mesh Terms:

KEYWORDS: Ploidies

MESH TERMS: genetics

Chemical & Substance for Abstract: The gene balance hypothesis: from classical genetics to modern genomics. Information

Substance Name:

Registry Number:

Grant and Affiliation Information for The gene balance hypothesis: from classical genetics to modern genomics.

AFFILIATION: Division of Biological Sciences, University of Missouri, Columbia, MO 65211, USA. birchlerj@missouri.edu

Country: United States

AGENCY: United States NIGMS

GRANT: R01 GM068042

ACRONYM: GM

MEDLINETA: Plant Cell

REFSOURCE:

DATABASENAME:

ACCESSION NUMBER:

Number Hits: 0

The gene balance hypothesis: from classical genetics to modern genomics Related Publications

• Amino Acid repeats and the structure and evolution of proteins.
• Differential aggregation and functional impairment induced by polyalanine expansions in FOXL2, a transcription factor involved in cranio-facial and ovarian development.
• Exploring the molecular etiology of dominant-negative mutations.
• FOXO3a variants in patients with premature ovarian failure.
• Mutations in the NOG gene are not a common cause of nonsyndromic premature ovarian failure.
• Preservation of genes involved in sterol metabolism in cholesterol auxotrophs: facts and hypotheses.
• Protein coevolution and isoexpression in yeast macromolecular complexes.
• The gene balance hypothesis: from classical genetics to modern genomics.
• The identification and characterization of a FOXL2 response element provides insights into the pathogenesis of mutant alleles.
• The mutations and potential targets of the forkhead transcription factor FOXL2.
• The post-translational modification profile of the forkhead transcription factor FOXL2 suggests the existence of parallel processive/concerted modification pathways.