Special Feature

User Panel

My Panel

My Panel

Bookmark Science Articles

Recent News
Bookmark / Share This Science Site

Pulmonary inflammation and bronchopulmonary dysplasia.

Pulmonary inflammation and bronchopulmonary dysplasia. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

  • Abstract Text of This Paper
  • Journal Published
  • MeSH Keywords of This Abstract
  • Chemicals and Substances Used in this Paper
  • Grants and Granting Agency of this Research
  • Database Accession Numbers Used in this Paper
  • Related Papers
  • Related Research Tags
  • Rate this Research Paper
  • Pulmonary inflammation and bronchopulmonary dysplasia. Abstract Text:

    c p speerC P Speer,c p speerC P Speer,

    Various pre- and postnatal risk factors, which act additively or synergistically induce an injurious inflammatory response in the airways and the pulmonary interstitium of preterm infants with bronchopulmonary dysplasia. This inflammatory response is characterized by an accumulation of neutrophils and macrophages as well as an arsenal of proinflammatory mediators that affect the endothelium and alveolar-capillary integrity. Besides proinflammatory cytokines and toxic oxygen radicals, lipid mediators as well as potent proteases may be responsible for acute lung injury. There is increasing evidence that an imbalance between pro- and anti-inflammatory factors, which should protect the alveoli and lung tissue, are key features in the pathogenesis of bronchopulmonary dysplasia. In addition, a subnormal generation of growth factors may affect alveolarization and vascular development in preterm infants with bronchopulmonary dysplasia. In this condensed review article, the current concepts on the possible role of inflammation in the evolution of bronchopulmonary dysplasia will be summarized.

    Pulmonary inflammation and bronchopulmonary dysplasia. Publishing Authors By Initials

    cp speerCP Speer,cp speerCP Speer,

    For similar abstracts research abstracts see: abstracts research

    PUBMED ID PMID:

    MEDLINE DATE:

    Pulmonary inflammation and bronchopulmonary dysplasia. Journal Published:

    PUBLICATION TYPE: Review

    Journal: Journal of perinatology : official journal of the

    VOLUME: 26 Suppl 1

    Page Numbers: S57-62; discussion S63-4

    Journal Abbreviation:

    ISSN: 0743-8346

    DAY: 20

    MONTH: May

    YEAR: 2006

    Pulmonary inflammation and bronchopulmonary dysplasia. Information

    Number of References: 44

    LANGUAGE: eng

    NlmUniqueID: 8501884

    Pulmonary inflammation and bronchopulmonary dysplasia. Keywords Mesh Terms:

    KEYWORDS:

    MESH TERMS:

    Chemical & Substance for Abstract: Pulmonary inflammation and bronchopulmonary dysplasia. Information

    Substance Name:

    Registry Number:

    Grant and Affiliation Information for Pulmonary inflammation and bronchopulmonary dysplasia.

    AFFILIATION: Department of Pediatrics, University Children's Hospital, Würzburg, Germany. speer_c@klinik.uni-wuerzburg.de

    Country: United States

    United States Research PublicationUnited States Research Publication

    AGENCY:

    GRANT:

    ACRONYM:

    MEDLINETA: J Perinatol

    REFSOURCE:

    DATABASENAME:

    ACCESSION NUMBER:

    Number Hits: 0

    Pulmonary inflammation and bronchopulmonary dysplasia Related Publications

     

    Molecular Station USER Menu

    Welcome to Molecular Station!

    You have to register before you can post on our forums or use our advanced features. Register Now! Its Free and Fast!

    Already registered? Login now below.

    User Name:

    Password:

    Already registered and Forgot your password? Click below to recover it.

    Recover Lost Password

    Join now - it's fast and free!

    Molecular Station is THE largest network of researchers, scientists and science lovers anywhere!

    Research Terms of Usage and Disclaimer
    Home
    Features

    Protocols

    DNA Forum

    Science Forum

    DNA Forum
    Biology Forum

    Science News


    [CaRP] XML error: Invalid document end at line 2

    For more click here:Science News