Potentially useful combinations of chemotherapy detected in mouse tumor systems.

Potentially useful combinations of chemotherapy detected in mouse tumor systems. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

Potentially useful combinations of chemotherapy detected in mouse tumor systems. Abstract Text:

Potentially useful combinations of chemotherapy detected in mouse tumor systems. Publishing Authors By Initials

For similar organic chemicals: triazenes research abstracts see: organic chemicals: triazenes research

PUBMED ID PMID:

MEDLINE DATE:

Potentially useful combinations of chemotherapy detected in mouse tumor systems. Journal Published:

PUBLICATION TYPE: Journal Article

Journal: Cancer chemotherapy reports. Part 2

VOLUME: 4

Page Numbers: 33-43

Journal Abbreviation:

ISSN: 0069-0120

DAY: 29

MONTH: Mar

YEAR: 1974

Potentially useful combinations of chemotherapy detected in mouse tumor systems. Information

Number of References:

LANGUAGE: eng

NlmUniqueID: 7607106

Potentially useful combinations of chemotherapy detected in mouse tumor systems. Keywords Mesh Terms:

KEYWORDS: Triazenes

MESH TERMS: therapeutic use

Chemical & Substance for Abstract: Potentially useful combinations of chemotherapy detected in mouse tumor systems. Information

Substance Name: Camptothecin

Registry Number: 7689-03-4

Grant and Affiliation Information for Potentially useful combinations of chemotherapy detected in mouse tumor systems.

AFFILIATION:

Country: UNITED STATES

AGENCY:

GRANT:

ACRONYM:

MEDLINETA: Cancer Chemother Rep 2

REFSOURCE:

DATABASENAME:

ACCESSION NUMBER:

Number Hits: 0

Potentially useful combinations of chemotherapy detected in mouse tumor systems Related Publications

• Activation of extracellular signal-regulated kinase and c-Jun-NH(2)-terminal kinase but not p38 mitogen-activated protein kinases is required for RRR-alpha-tocopheryl succinate-induced apoptosis of human breast cancer cells.
• Body temperature fluctuations in the periparturient horse mare.
• Differential response of human ovarian cancer cells to induction of apoptosis by vitamin E Succinate and vitamin E analogue, alpha-TEA.
• Effects of RRR-alpha-tocopheryl succinate on IL-1 and PGE2 production by macrophages.
• Elevated ankyrin G in a plexiform neurofibroma and neuromas associated with pain.
• Equine body temperature and progesterone fluctuations during estrus and near parturition.
• Evidence for role of transforming growth factor-beta in RRR-alpha-tocopheryl succinate-induced apoptosis of human MDA-MB-435 breast cancer cells.
• Involvement of activator protein-1 (AP-1) in induction of apoptosis by vitamin E succinate in human breast cancer cells.
• Modulation of murine EL-4 thymic lymphoma cell proliferation and cytokine production by vitamin E succinate.
• Pro-apoptotic mechanisms of action of a novel vitamin E analog (alpha-TEA) and a naturally occurring form of vitamin E (delta-tocotrienol) in MDA-MB-435 human breast cancer cells.
• RRR-alpha-tocopheryl succinate enhances TGF-beta 1, -beta 2, and -beta 3 and TGF-beta R-II expression by human MDA-MB-435 breast cancer cells.
• RRR-alpha-tocopheryl succinate induced interleukin-2 production by avian splenic T lymphocytes and murine EL-4 thymic lymphoma cells.
• RRR-alpha-tocopheryl succinate induces MDA-MB-435 and MCF-7 human breast cancer cells to undergo differentiation.
• RRR-alpha-tocopheryl succinate induces apoptosis in avian retrovirus-transformed lymphoid cells.
• RRR-alpha-tocopheryl succinate induction of DNA synthesis arrest of human MDA-MB-435 cells involves TGF-beta-independent activation of p21Waf1/Cip1.
• RRR-alpha-tocopheryl succinate induction of prolonged activation of c-jun amino-terminal kinase and c-jun during induction of apoptosis in human MDA-MB-435 breast cancer cells.
• RRR-alpha-tocopheryl succinate inhibits DNA synthesis and enhances the production and secretion of biologically active transforming growth factor-beta by avian retrovirus-transformed lymphoid cells.
• RRR-alpha-tocopheryl succinate inhibits EL4 thymic lymphoma cell growth by inducing apoptosis and DNA synthesis arrest.
• RRR-alpha-tocopheryl succinate inhibits proliferation and enhances secretion of transforming growth factor-beta (TGF-beta) by human breast cancer cells.
• RRR-alpha-tocopheryl succinate inhibits the proliferation of human prostatic tumor cells with defective cell cycle/differentiation pathways.
• RRR-alpha-tocopheryl succinate modulation of human promyelocytic leukemia (HL-60) cell proliferation and differentiation.
• RRR-alpha-tocopheryl succinate-induced apoptosis of human breast cancer cells involves Bax translocation to mitochondria.
• Role of extracellular signal-regulated kinase pathway in RRR-alpha-tocopheryl succinate-induced differentiation of human MDA-MB-435 breast cancer cells.
• Telemetric monitoring of body temperature in the horse mare.
• Uterine electromyographic activity in horse mares as measured by radiotelemetry.
• Vitamin E succinate (VES) induces Fas sensitivity in human breast cancer cells: role for Mr 43,000 Fas in VES-triggered apoptosis.
• Vitamin E succinate induces apoptosis in human prostate cancer cells: role for Fas in vitamin E succinate-triggered apoptosis.
• Vitamin E succinate induction of HL-60 cell adhesion: a role for fibronectin and a 72-kDa fibronectin-binding molecule.
• alpha-TEA inhibits survival and enhances death pathways in cisplatin sensitive and resistant human ovarian cancer cells.
• c-Jun involvement in vitamin E succinate induced apoptosis of reticuloendotheliosis virus transformed avian lymphoid cells.