Oxidative stress, endothelial dysfunction and cerebrovascular disease.

Oxidative stress, endothelial dysfunction and cerebrovascular disease. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

Oxidative stress, endothelial dysfunction and cerebrovascular disease. Abstract Text:

Guillermo Zalba,Ana ,Gorka , Moreno,Oscar Beloqui,Javier ,Guillermo Zalba,Ana ,Gorka , Moreno,Oscar Beloqui,Javier ,

Endothelial dysfunction is a marker of atherosclerosis and contributes to the atherogenic process and the development of atherothrombotic complications. Oxidative stress has been implicated in the development of endothelial dysfunction through alterations of the nitric oxide metabolism. A number of evidence suggests a role for phagocytic-cell-mediated oxidative stress in diminished nitric oxide availability that is present in patients with atherosclerotic risk factors such as arterial hypertension. Thus, the combination of an excessive production of reactive oxygen species, namely superoxide anion, with an impaired antioxidant defense capacity leading to oxidative stress may facilitate the development and progression of atherosclerosis. Findings from recent clinical studies suggest that this mechanism can be operative in patients with cerebrovascular disease. This view may increase our capabilities to understand the pathophysiology of cerebrovascular disease, as well as to stimulate the design of new therapeutic strategies aimed to prevent and control the atherosclerotic process in patients presenting this condition.

Oxidative stress, endothelial dysfunction and cerebrovascular disease. Publishing Authors By Initials

G Zalba,A ,G ,MU Moreno,O Beloqui,J ,G Zalba,A ,G ,MU Moreno,O Beloqui,J ,

For similar abstracts research abstracts see: abstracts research

PUBMED ID PMID:

MEDLINE DATE:

Oxidative stress, endothelial dysfunction and cerebrovascular disease. Journal Published:

PUBLICATION TYPE: Journal Article

Journal: Cerebrovascular diseases (Basel, Switzerland)

VOLUME: 24 Suppl 1

Page Numbers: 24-9

Journal Abbreviation: Cerebrovasc. Dis.

ISSN: 1015-9770

DAY: 1

MONTH: 11

YEAR: 2007

Oxidative stress, endothelial dysfunction and cerebrovascular disease. Information

Number of References:

LANGUAGE: eng

NlmUniqueID: 9100851

Oxidative stress, endothelial dysfunction and cerebrovascular disease. Keywords Mesh Terms:

KEYWORDS:

MESH TERMS:

Chemical & Substance for Abstract: Oxidative stress, endothelial dysfunction and cerebrovascular disease. Information

Substance Name:

Registry Number:

Grant and Affiliation Information for Oxidative stress, endothelial dysfunction and cerebrovascular disease.

AFFILIATION: Division of Cardiovascular Sciences, Centre for Applied Medical Research, Pamplona, Spain.

Country: Switzerland

AGENCY:

GRANT:

ACRONYM:

MEDLINETA: Cerebrovasc Dis

REFSOURCE:

DATABASENAME:

ACCESSION NUMBER:

Number Hits: 0

Oxidative stress, endothelial dysfunction and cerebrovascular disease Related Publications

• A solid-phase extraction and size-exclusion liquid chromatographic method for polyethylene glycol 25 p-aminobenzoic acid determination in urine: Validation for urinary excretion studies of users of sunscreens.
• AT1 receptor antagonism attenuates target organ effects of salt excess in SHRs without affecting pressure.
• Acute and one-year effects of cinacalcet in patients with persistent primary hyperparathyroidism after unsuccessful parathyroidectomy.
• Antithrombotic therapy and the transition to the catheterization laboratory in UA/NSTEMI.
• Apoptosis in hypertensive heart disease: a clinical approach.
• Association of depressed cardiac gp130-mediated antiapoptotic pathways with stimulated cardiomyocyte apoptosis in hypertensive patients with heart failure.
• Bovine early embryonic development and vitamin a.
• Detection of fractional steps in cargo movement by the collective operation of kinesin-1 motors.
• Detection of the CHEK2 1100delC mutation by MLPA BRCA1/2 analysis: a worthwhile strategy for its clinical applicability in 1100delC low-frequency populations?
• Determination of ellagic acid in oak leaves and in sheep ruminal fluid by ion-pair RP-HPLC.
• Effect of short-duration electrically induced tachycardia on regional myocardial contractility.
• Feature extraction and selection for the automatic detection of hard exudates in retinal images.
• Identification of PatL1, a human homolog to yeast P body component Pat1.
• Identification of the spI products of Balbiani ring genes in Chironomus thummi.
• Impact of collagen type I turnover on the long-term response to cardiac resynchronization therapy.
• Influence of thyroid dysfunction on serum concentrations of adipocytokines.
• Oxidative stress, endothelial dysfunction and cerebrovascular disease.
• Radiologic evaluation of the Llullaillaco mummies.
• Results of biliopancreatic diversion after five years.
• Saccharomyces cerevisiae: a versatile eukaryotic system in virology.
• Solid organ transplantation and response to vaccination.
• Sulphur poisoning of palladium catalysts used for methane combustion: Effect of the support.
• Upregulation of myocardial Annexin A5 in hypertensive heart disease: association with systolic dysfunction.
• [Are lumbar pains in workers occupational diseases of diseases?]
• [Not Available.]
• [Not Available.]
• [Not Available.]
• [Results of the quantitative frog test in clinical routine HCG determinations in the serum]
• [Role of Spanish neurology in knowledge about headaches with focal neurological deficit and lymphocytic pleocytosis.]
• [Therapeutic possibilities in patients with selective pituitary resistance to thyroid hormones.]