Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy.

Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy. Research Abstract Details 

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Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy. Abstract Text:

Neutrophil chemoattractants cause neutrophil-dependent goblet cell degranulation in guinea pig and human airways through a novel mechanism involving close contact between neutrophils and goblet cells, mediated by neutrophil elastase. The chemoattractant plays various roles in the response: It causes neutrophil recruitment and mobilizes elastase to the surface of the neutrophil. In addition, it induces surface expression of adhesion molecules, which allow the neutrophil to interact with the goblet cell surface, resulting in goblet cell secretion.

Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy. Publishing Authors By Initials

For similar cells: blood cells: leukocytes: granulocytes: neutrophils research abstracts see: cells: blood cells: leukocytes: granulocytes: neutrophils research

PUBMED ID PMID:

MEDLINE DATE:

Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy. Journal Published:

PUBLICATION TYPE: Research Support, U.S. Gov't,

Journal: Pediatric pulmonology. Supplement

VOLUME: 18

Page Numbers: 54-5

Journal Abbreviation:

ISSN: 1054-187X

DAY: 26

MONTH: 02

YEAR: 1999

Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy. Information

Number of References:

LANGUAGE: eng

NlmUniqueID: 9014095

Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy. Keywords Mesh Terms:

KEYWORDS: Neutrophils

MESH TERMS: physiology

Chemical & Substance for Abstract: Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy. Information

Substance Name: Leukocyte Elastase

Registry Number: EC 3.4.21.37

Grant and Affiliation Information for Mechanisms of hypersecretion in acute asthma, proposed cause of death, and novel therapy.

AFFILIATION: Cardiovascular Research Institute, University of California, San Francisco 94143-0130, USA. janadel@itsa.ucsf.edu

Country: UNITED STATES

AGENCY: United States NHLBI

GRANT: PPG HL-24136

ACRONYM: HL

MEDLINETA: Pediatr Pulmonol Suppl

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