Special Feature

User Panel

My Panel

My Panel

Bookmark Science Articles

Recent News
Bookmark / Share This Science Site

Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury.

Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

  • Abstract Text of This Paper
  • Journal Published
  • MeSH Keywords of This Abstract
  • Chemicals and Substances Used in this Paper
  • Grants and Granting Agency of this Research
  • Database Accession Numbers Used in this Paper
  • Related Papers
  • Related Research Tags
  • Rate this Research Paper
  • Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury. Abstract Text:

    r e beamishR E Beamish,

    Oxidant injury contributes to myocardial stunning, and cardiac ischemic and reperfusion injury. Vitamin E is the major--and perhaps the only--lipid soluble, chain-breaking antioxidant in the heart. Vitamin E and its analogues potentially offer significant advantages for the prevention of ischemic and reperfusion injury. Recent investigations have suggested that modified vitamin E analogues may be more efficacious than vitamin E and may permit myocardial salvage from acute myocardial ischemic injury.

    Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury. Publishing Authors By Initials

    re beamishRE Beamish,

    For similar heterocyclic compounds: heterocyclic compounds, 2-ring: benzopyrans: vitamin e research abstracts see: heterocyclic compounds: heterocyclic compounds, 2-ring: benzopyrans: vitamin e research

    PUBMED ID PMID:

    MEDLINE DATE:

    Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury. Journal Published:

    PUBLICATION TYPE: Review

    Journal: The Canadian journal of cardiology

    VOLUME: 9

    Page Numbers: 89-93

    Journal Abbreviation:

    ISSN: 1916-7075

    DAY: 3

    MONTH: 04

    YEAR: 2008

    Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury. Information

    Number of References: 19

    LANGUAGE: eng

    NlmUniqueID: 8510280

    Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury. Keywords Mesh Terms:

    KEYWORDS: Vitamin E

    MESH TERMS: therapeutic use

    Chemical & Substance for Abstract: Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury. Information

    Substance Name: 6-hydroxy-2,5,7,8-tetramethylchroman-2-c

    Registry Number: 56305-04-5

    Grant and Affiliation Information for Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury.

    AFFILIATION: Department of Clinical Biochemistry, Toronto Hospital, Ontario.

    Country: CANADA

    CANADA Research PublicationCANADA Research Publication

    AGENCY:

    GRANT:

    ACRONYM:

    MEDLINETA: Can J Cardiol

    REFSOURCE: Can J Cardiol. 1993 Jan-Feb;9(1):29-31

    DATABASENAME:

    ACCESSION NUMBER:

    Number Hits: 0

    Future directions of vitamin E and its analogues in minimizing myocardial ischemia-reperfusion injury Related Publications

     

    Molecular Station USER Menu

    Welcome to Molecular Station!

    You have to register before you can post on our forums or use our advanced features. Register Now! Its Free and Fast!

    Already registered? Login now below.

    User Name:

    Password:

    Already registered and Forgot your password? Click below to recover it.

    Recover Lost Password

    Join now - it's fast and free!

    Molecular Station is THE largest network of researchers, scientists and science lovers anywhere!

    Research Terms of Usage and Disclaimer
    Home
    Features

    Protocols

    DNA Forum

    Science Forum

    DNA Forum
    Biology Forum

    Science News


    [CaRP] XML error: Invalid document end at line 2

    For more click here:Science News