Enhanced emotion-induced amnesia in borderline personality disorder.

Enhanced emotion-induced amnesia in borderline personality disorder. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

Enhanced emotion-induced amnesia in borderline personality disorder. Abstract Text:

Hurlemann,Barbara Hawellek,Wolfgang Maier,Raymond J Dolan, Hurlemann,Barbara Hawellek,Wolfgang Maier,Raymond J Dolan,

BACKGROUND: Current biological concepts of borderline personality disorder (BPD) emphasize the interference of emotional hyperarousal and cognitive functions. A prototypical example is episodic memory. Pre-clinical investigations of emotion-episodic memory interactions have shown specific retrograde and anterograde episodic memory changes in response to emotional stimuli. These changes are amygdala dependent and vary as a function of emotional arousal and valence. METHOD: To determine whether there is amygdala hyper-responsiveness to emotional stimuli as the underlying pathological substrate of cognitive dysfunction in BPD, 16 unmedicated female patients with BPD were tested on the behavioural indices of emotion-induced amnesia and hypermnesia established in 16 healthy controls. RESULTS: BPD patients displayed enhanced retrograde and anterograde amnesia in response to presentation of negative stimuli, while positive stimuli elicited no episodic memory-modulating effects. CONCLUSION: These findings suggest that an amygdala hyper-responsiveness to negative stimuli may serve as a crucial aetiological contributor to emotion-induced cognitive dysfunction in BPD.

Enhanced emotion-induced amnesia in borderline personality disorder. Publishing Authors By Initials

R Hurlemann,B Hawellek,W Maier,RJ Dolan,R Hurlemann,B Hawellek,W Maier,RJ Dolan,

For similar abstracts research abstracts see: abstracts research

PUBMED ID PMID:

MEDLINE DATE:

Enhanced emotion-induced amnesia in borderline personality disorder. Journal Published:

PUBLICATION TYPE: Research Support, Non-U.S. Gov

Journal: Psychological medicine

VOLUME: 37

Page Numbers: 971-81

Journal Abbreviation:

ISSN: 0033-2917

DAY: 16

MONTH: 01

YEAR: 2007

Enhanced emotion-induced amnesia in borderline personality disorder. Information

Number of References:

LANGUAGE: eng

NlmUniqueID: 1254142

Enhanced emotion-induced amnesia in borderline personality disorder. Keywords Mesh Terms:

KEYWORDS:

MESH TERMS:

Chemical & Substance for Abstract: Enhanced emotion-induced amnesia in borderline personality disorder. Information

Substance Name:

Registry Number:

Grant and Affiliation Information for Enhanced emotion-induced amnesia in borderline personality disorder.

AFFILIATION: Department of Psychiatry, University of Bonn, Bonn, Germany. rene.hurlemann@ukb.uni-bonn.de

Country: England

AGENCY:

GRANT:

ACRONYM:

MEDLINETA: Psychol Med

REFSOURCE:

DATABASENAME:

ACCESSION NUMBER:

Number Hits: 0

Enhanced emotion-induced amnesia in borderline personality disorder Related Publications

• 'That's just the cesspool where they dump all the trash': exploring working class men's perceptions and experiences of social capital and health.
• A genome-wide approach to identify genetic variants that contribute to etoposide-induced cytotoxicity.
• Cholinesterase inhibition modulates visual and attentional brain responses in Alzheimer's disease and health.
• Differential ethylene sensitivity of epidermal cells is involved in the establishment of cell pattern in the Arabidopsis root.
• Distinct and convergent visual processing of high and low spatial frequency information in faces.
• Electromotile hearing: acoustic tones mask psychophysical response to high-frequency electrical stimulation of intact guinea pig cochleae.
• Emotion-induced retrograde amnesia varies as a function of noradrenergic-glucocorticoid activity.
• Enhanced emotion-induced amnesia in borderline personality disorder.
• Exploring efferent-mediated DPOAE adaptation in three different guinea pig strains.
• Finding value. ACHE provides exceptional services and benefits to its members.
• Gastrointestinal staple line reinforcement.
• Genetic association and brain morphology studies and the chromosome 8p22 pericentriolar material 1 (PCM1) gene in susceptibility to schizophrenia.
• Human pavlovian instrumental transfer.
• Identification of genetic variants contributing to cisplatin-induced cytotoxicity by use of a genomewide approach.
• Increased burst pressure in gastrointestinal staple-lines using reinforcement with a bioprosthetic material.
• Instability in the diagnosis of metabolic syndrome in adolescents.
• Keynote address: revaluing the orbital prefrontal cortex.
• Mapping genes that contribute to daunorubicin-induced cytotoxicity.
• Mesenteric artery occlusion secondary to activated protein C resistance: a life-threatening combination.
• Molecular mechanisms of resistance and toxicity associated with platinating agents.
• Phase I trial of polifeprosan 20 with carmustine implant plus continuous infusion of intravenous O6-benzylguanine in adults with recurrent malignant glioma: new approaches to brain tumor therapy CNS consortium trial.
• Role of MGMT in protecting against cyclophosphamide-induced toxicity in cells and animals.
• Role of O6-methylguanine-DNA methyltransferase in protecting from alkylating agent-induced toxicity and mutations in mice.
• Role of features and second-order spatial relations in face discrimination, face recognition, and individual face skills: behavioral and functional magnetic resonance imaging data.
• Social regulation of affective experience of humor.
• Soul searching: a brief history of the mind/body debate in the neurosciences.
• Sowing the seeds of dialogue: public engagement through plant science.
• Tyr346-->Cys mutation results in factor VIII:C assay discrepancy and a normal bleeding phenotype - is this mild haemophilia A?
• University of Florida College of Dentistry's "Case of the Month": Evaluating a web-based continuing dental education course for clinical oral pathology.
• Vagus nerve stimulation for treatment-resistant depression: behavioral and neural effects on encoding negative material.