Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis.

Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis. Research Abstract Details 

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Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis. Abstract Text:

M Ceci,P Gallo,M Santonastasi,S Grimaldi,M V G Latronico,A Pitisci,E Missol-Kolka,M C Scimia,D Catalucci,D Hilfiker-Kleiner,G Condorelli,

Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis. Publishing Authors By Initials

M Ceci,P Gallo,M Santonastasi,S Grimaldi,MV Latronico,A Pitisci,E Missol-Kolka,MC Scimia,D Catalucci,D Hilfiker-Kleiner,G Condorelli,

For similar peptides: intercellular signaling peptides and proteins: angiogenic proteins: vascular endothelial growth factors: vascular endothelial growth factor a research abstracts see: peptides: intercellular signaling peptides and proteins: angiogenic proteins: vascular endothelial growth factors: vascular endothelial growth factor a research

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Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis. Journal Published:

PUBLICATION TYPE: Research Support, Non-U.S. Gov

Journal: Cell death and differentiation

VOLUME: 14

Page Numbers: 1060-2

Journal Abbreviation:

ISSN: 1350-9047

DAY: 19

MONTH: 01

YEAR: 2007

Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis. Information

Number of References:

LANGUAGE: eng

NlmUniqueID: 9437445

Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis. Keywords Mesh Terms:

KEYWORDS: Vascular Endothelial Growth Factor A

MESH TERMS: metabolism

Chemical & Substance for Abstract: Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis. Information

Substance Name: Proto-Oncogene Proteins c-akt

Registry Number: EC 2.7.1.37

Grant and Affiliation Information for Cardiac-specific overexpression of E40K active Akt prevents pressure overload-induced heart failure in mice by increasing angiogenesis and reducing apoptosis.

AFFILIATION:

Country: England

AGENCY: United States NHLBI

GRANT: R01 HL078797-01A1

ACRONYM: HL

MEDLINETA: Cell Death Differ

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