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Calmodulin-binding transcription activator (CAMTA) 3 mediates biotic defense responses in Arabidopsis.

Calmodulin-binding transcription activator (CAMTA) 3 mediates biotic defense responses in Arabidopsis. Research Abstract Details 

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  • Calmodulin-binding transcription activator (CAMTA) 3 mediates biotic defense responses in Arabidopsis. Abstract Text:

    Calmodulin-binding transcription activator (CAMTA) 3 (also called SR1) is a calmodulin-binding transcription factor in Arabidopsis. Two homozygous T-DNA insertion mutants (camta3-1, camta3-2) showed enhanced spontaneous lesions. Transcriptome analysis of both mutants revealed 6 genes with attenuated expression and 99 genes with elevated expression. Of the latter, 32 genes are related to defense against pathogens (e.g. WRKY33, PR1 and chitinase). Propagation of a virulent strain of the bacterial pathogen Pseudomonas syringae and the fungal pathogen Botrytis cinerea were attenuated in both mutants. Moreover, both mutants accumulated high levels of H(2)O(2). We suggest that CAMTA3 regulates the expression of a set of genes involved in biotic defense responses.

    Calmodulin-binding transcription activator (CAMTA) 3 mediates biotic defense responses in Arabidopsis. Publishing Authors By Initials

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    Calmodulin-binding transcription activator (CAMTA) 3 mediates biotic defense responses in Arabidopsis. Journal Published:

    PUBLICATION TYPE: Journal Article

    Journal: FEBS letters

    VOLUME: 582

    Page Numbers: 943-8

    Journal Abbreviation: FEBS Lett.

    ISSN: 0014-5793

    DAY: 25

    MONTH: 02

    YEAR: 2008

    Calmodulin-binding transcription activator (CAMTA) 3 mediates biotic defense responses in Arabidopsis. Information

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    LANGUAGE: eng

    NlmUniqueID: 155157

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    Grant and Affiliation Information for Calmodulin-binding transcription activator (CAMTA) 3 mediates biotic defense responses in Arabidopsis.

    AFFILIATION: Department of Plant Sciences, Faculty of Life Sciences, Tel Aviv University, Tel Aviv 69978, Israel.

    Country: Netherlands

    Netherlands Research PublicationNetherlands Research Publication

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    MEDLINETA: FEBS Lett

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