-Jon E Wergedal Researcher Activity Profile

Research Author Detailed Information 

Jon E Wergedal Publication Rate By Year

Jon E Wergedal has published 1 paper(s) in 1982, 1 paper(s) in 1984, 1 paper(s) in 1987, 1 paper(s) in 1990, 1 paper(s) in 2003, 2 paper(s) in 2006, 13 paper(s) in 2007, for a total of 20 research publications in total.

Jon E Je Wergedal Author Information

LAST NAME: wergedal

FIRST NAME: Jon E

INITIALS: je

AFFILIATION:

Papers

Jon E Wergedal's Publication Record

1. Identification of mouse Duffy antigen receptor for chemokines (Darc) as a BMD QTL gene. Year Published: 2007
Musculoskeletal Disease Center, Jerry L. Pettis Memorial VA Medical Center, and Department of Medicine and Biochemistry, Loma Linda University, California 92357, USA.
2. New quantitative trait loci that regulate wound healing in an intercross progeny from DBA/1J and 129 x 1/SvJ inbred strains of mice. Year Published: 2006
Musculoskeletal Disease Center, Jerry L. Pettis Memorial Veterans Administration Medical Center, Loma Linda, CA 92357, USA.
3. Pregnancy-associated plasma protein-A increases osteoblast proliferation in vitro and bone formation in vivo. Year Published: 2006
Musculoskeletal Disease Center, J. L. Pettis Veterans Affairs Medical Center (151), 11201 Benton Street, and Department of Medicine, Loma Linda University, California 92357, USA. Xuezhong.Qin@med.va.gov
4. TNF-related weak inducer of apoptosis (TWEAK) is a potent skeletal muscle-wasting cytokine. Year Published: 2007
Musculoskeletal Disease Center, Jerry L. Pettis Memorial Veterans Administration Medical Center, Loma Linda, CA 92357, USA.
5. Mapping genetic loci that regulate lipid levels in a NZB/B1NJxRF/J intercross and a combined intercross involving NZB/B1NJ, RF/J, MRL/MpJ, and SJL/J mouse strains. Year Published: 2007
Musculoskeletal Disease Center, Loma Linda VA Health Care Systems, Loma Linda, CA, USA.
6. Genetic regulation of femoral bone mineral density: complexity of sex effect in chromosome 1 revealed by congenic sublines of mice. Year Published: 2007
Musculoskeletal Disease Center, Jerry L. Pettis Memorial VA Medical Center, 11201 Benton Street, and Department of Biochemistry, Loma Linda University, CA 92357, USA.
7. Disruption of insulin-like growth factor-I expression in type IIalphaI collagen-expressing cells reduces bone length and width in mice. Year Published: 2007
Jerry L. Pettis Veterans Affairs Medical Center and Loma Linda University, Loma Linda, California 92357, USA.
8. Sca-1(+) hematopoietic cell-based gene therapy with a modified FGF-2 increased endosteal/trabecular bone formation in mice. Year Published: 2007
Musculoskeletal Disease Center, Jerry L. Pettis Memorial V.A. Medical Center, Loma Linda, California 92357, USA.
9. Chemical mutagenesis induced two high bone density mouse mutants map to a concordant distal chromosome 4 locus. Year Published: 2007
Musculoskeletal Disease Center (151), Loma Linda VA Healthcare Systems, Loma Linda, CA 92357, USA.
10. Conditional Deletion of Insulin-Like Growth Factor-I in Collagen Type 1{alpha}2-Expressing Cells Results in Postnatal Lethality and a Dramatic Reduction in Bone Accretion. Year Published: 2007
Musculoskeletal Disease Center (151), Jerry L. Pettis Memorial Veterans Affairs Medical Center, 11201 Benton Street, Loma Linda, California 92357. subburaman.mohan@va.gov.
11. Transgenic overexpression of pregnancy-associated plasma protein-a increases the somatic growth and skeletal muscle mass in mice. Year Published: 2007
Musculoskeletal Disease Center, J. L. Pettis Veterans Affairs Medical Center (151), 11201 Benton Street, Loma Linda, California 92357. xuezhong.qin@med.va.gov.
12. Conditional deletion of insulin-like growth factor-I in collagen type 1alpha2-expressing cells results in postnatal lethality and a dramatic reduction in bone accretion. Year Published: 2007
Musculoskeletal Disease Center, Jerry L. Pettis Memorial Veterans Affairs Medical Center, 11201 Benton Street, Loma Linda, CA 92357, USA.
13. Transgenic overexpression of pregnancy-associated plasma protein-A increases the somatic growth and skeletal muscle mass in mice. Year Published: 2007
Musculoskeletal Disease Center, J. L. Pettis Veterans Affairs Medical Center (151), 11201 Benton Street, Loma Linda, CA 92357, USA.
14. A missense mutation in the mouse Col2a1 gene causes spondyloepiphyseal dysplasia congenita, hearing loss, and retinoschisis. Year Published: 2003
The Jackson Laboratory, Bar Harbor, Maine 04609, USA. lrd@jax.org
15. Sca-1(+) hematopoietic cell-based gene therapy with a modified FGF-2 increased endosteal/trabecular bone formation in mice. Year Published: 2007
Musculoskeletal Disease Center, Jerry L. Pettis Memorial V.A. Medical Center, Loma Linda, California 92357, USA.
16. Human skeletal growth factor: characterization of the mitogenic effect on bone cells in vitro. Year Published: 1982
17. Characterization of cells isolated and cultured from human bone. Year Published: 1984
18. Skeletal growth factor is produced by human osteoblast-like cells in culture. Year Published: 1987
19. Skeletal growth factor and other growth factors known to be present in bone matrix stimulate proliferation and protein synthesis in human bone cells. Year Published: 1990
Department of Medicine, Loma Linda University, CA.
20. Chemical mutagenesis induced two high bone density mouse mutants map to a concordant distal chromosome 4 locus. Year Published: 2007
Musculoskeletal Disease Center (151), Loma Linda VA Healthcare Systems, Loma Linda, CA 92357, USA.