-Bruce D Walker Researcher Activity Profile

Research Author Detailed Information 

Bruce D Walker Publication Rate By Year

Bruce D Walker has published 1 paper(s) in 1997, 1 paper(s) in 2003, 2 paper(s) in 2005, 29 paper(s) in 2007, 8 paper(s) in 2008, for a total of 41 research publications in total.

Bruce D Bd Walker Author Information

LAST NAME: walker

FIRST NAME: Bruce D

INITIALS: bd

AFFILIATION:

Papers

Bruce D Walker's Publication Record

1. Extensive intrasubtype recombination in South African human immunodeficiency virus type 1 subtype C infections. Year Published: 2007
Department of Microbiology, University of Washington, 1959 NE Pacific Street, Box 358070, Seattle, WA 98195-8070, USA. cmr@u.washington.edu
2. Characterization of full-length hepatitis C virus genotype 4 sequences. Year Published: 2007
Partners AIDS Research Center, Infectious Disease Division, Massachusetts General Hospital, Boston, MA, USA.
3. Suppression of human immunodeficiency virus type 1 replication by CD8+ cells: evidence for HLA class I-restricted triggering of cytolytic and noncytolytic mechanisms. Year Published: 1997
AIDS Research Center, Massachusetts General Hospital and Harvard Medical School, Charlestown 02129, USA.
4. [Specific immune responses to human immunodeficiency virus type 1 Gag, Tat, Rev and Nef proteins] Year Published: 2005
Center of Diagnosis and Treatment for Infectious Diseases of Chinese PLA, Tangdu Hospital, Fourth Military Medical University, Xi'an 710038, China.
5. Mutually exclusive T-cell receptor induction and differential susceptibility to human immunodeficiency virus type 1 mutational escape associated with a two-amino-acid difference between HLA class I subtypes. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, 149 13th Street, Room 5212, Charlestown, MA 02129, USA.
6. Identification of HLA-A11-restricted HIV-1-specific cytotoxic T-lymphocyte epitopes in China. Year Published: 2007
Department of Infectious Diseases, Tangdu Hospital Affiliated to the Fourth Military Medical University, Xi'an 710038, PR China.
7. Inter-clade cross-reactivity of HIV-1-specific T cell responses in human immunodeficiency virus type 1 infection in China. Year Published: 2007
Department of Infectious Diseases, Tangdu Hospital Affiliated to the Fourth Military Medical University, Xi'an, PR China.
8. Recognition of a defined region within p24 gag by CD8+ T cells during primary human immunodeficiency virus type 1 infection in individuals expressing protective HLA class I alleles. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02129, USA.
9. Pathways utilized by dendritic cells for binding, uptake, processing and presentation of antigens derived from HIV-1. Year Published: 2007
Department of Medicine and Pathology, School of Medicine, New York University, NY, USA.
10. Compensatory mutation partially restores fitness and delays reversion of escape mutation within the immunodominant HLA-B*5703-restricted Gag epitope in chronic human immunodeficiency virus type 1 infection. Year Published: 2007
Department of Pediatrics, University of Oxford, Peter Medawar Building for Pathogen Research, South Parks Rd., Oxford OX1 3SY, United Kingdom.
11. Immunologic evidence for lack of heterologous protection following resolution of HCV in patients with non-genotype 1 infection. Year Published: 2007
Partners AIDS Research Center, Infectious Disease Division, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA. julianszw@gmail.com
12. Extensive HLA class I allele promiscuity among viral CTL epitopes. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.
13. Human immunodeficiency virus controllers: mechanisms of durable virus control in the absence of antiretroviral therapy. Year Published: 2007
University of California, San Francisco, and San Francisco General Hospital, San Francisco, CA 94110, USA.
14. Human immunodeficiency virus type 1 specific cytotoxic T lymphocyte responses in Chinese infected with HIV-1 B'/C Recombinant (CRF07_BC). Year Published: 2007
State Key Laboratory for Infectious Disease Control and Prevention, National Center for AIDS/STD Control and Prevention, Chinese Center for Disease Control and Prevention, Beijing 100050, China. jping_chen@chinaaids.cn
15. Upregulation of CTLA-4 by HIV-specific CD4(+) T cells correlates with disease progression and defines a reversible immune dysfunction. Year Published: 2007
[1] Partners AIDS Research Center, Massachusetts General Hospital and Division of AIDS, Harvard Medical School, Boston, Massachusetts 02115, USA. [2] These authors contributed equally to this work.
16. Escape and compensation from early HLA-B57-mediated cytotoxic T-lymphocyte pressure on human immunodeficiency virus type 1 Gag alter capsid interactions with cyclophilin A. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital-East, CNY 6625, Charlestown, MA 02129, USA.
17. Random T-cell receptor recruitment in human immunodeficiency virus type 1 (HIV-1)-specific CD8+ T cells from genetically identical twins infected with the same HIV-1 strain. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Charlestown, MA 02129, USA.
18. Portable flanking sequences modulate CTL epitope processing. Year Published: 2007
Partners AIDS Research Center and Howard Hughes Medical Institute, Massachusetts General Hospital (MGH), Harvard Medical School, Boston, Massachusetts 02129, USA. sylvie_legall@hms.harvard.edu
19. Increased sequence diversity coverage improves detection of HIV-specific T cell responses. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02129, USA.
20. A viral CTL escape mutation leading to immunoglobulin-like transcript 4-mediated functional inhibition of myelomonocytic cells. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, and Harvard University Center for AIDS Research, Boston, MA 02129, USA.
21. Human immunodeficiency virus-specific CD8+ T-cell activity is detectable from birth in the majority of in utero-infected infants. Year Published: 2007
HIV Pathogenesis Programme, Doris Duke Medical Research Institute, University of KwaZulu-Natal, Durban, South Africa. Thobakgalec@ukzn.ac.za
22. Escape and compensation from early HLA-B57-mediated cytotoxic T-lymphocyte pressure on human immunodeficiency virus type 1 Gag alter capsid interactions with cyclophilin A. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital-East, CNY 6625, Charlestown, MA 02129, USA.
23. Random T-cell receptor recruitment in human immunodeficiency virus type 1 (HIV-1)-specific CD8+ T cells from genetically identical twins infected with the same HIV-1 strain. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Charlestown, MA 02129, USA.
24. A viral CTL escape mutation leading to immunoglobulin-like transcript 4-mediated functional inhibition of myelomonocytic cells. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, and Harvard University Center for AIDS Research, Boston, MA 02129, USA.
25. Human immunodeficiency virus-specific CD8+ T-cell activity is detectable from birth in the majority of in utero-infected infants. Year Published: 2007
HIV Pathogenesis Programme, Doris Duke Medical Research Institute, University of KwaZulu-Natal, Durban, South Africa. Thobakgalec@ukzn.ac.za
26. Upregulation of CTLA-4 by HIV-specific CD4+ T cells correlates with disease progression and defines a reversible immune dysfunction. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA. dkaufmann@partners.org
27. Increased sequence diversity coverage improves detection of HIV-specific T cell responses. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02129, USA.
28. Broad and Gag-Biased HIV-1 Epitope Repertoires Are Associated with Lower Viral Loads. Year Published: 2008
Department of Microbiology, University of Washington School of Medicine, Seattle, Washington, United States of America.
29. Upregulation of CTLA-4 by HIV-specific CD4+ T cells correlates with disease progression and defines a reversible immune dysfunction. Year Published: 2007
Partners AIDS Research Center, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts 02115, USA. dkaufmann@partners.org
30. CCL3L1 and CCR5 influence cell-mediated immunity and affect HIV-AIDS pathogenesis via viral entry-independent mechanisms. Year Published: 2007
Infectious Disease Clinical Research Program, Wilford Hall United States Air Force Medical Center, Lackland Air Force Base, Texas 78236, USA. mdolan@hjf.org
31. CD161 expression on hepatitis C virus-specific CD8+ T cells suggests a distinct pathway of T cell differentiation. Year Published: 2008
Peter Medawar Building for Pathogen Research, University of Oxford, Oxford, UK.
32. Compensatory mutation partially restores fitness and delays reversion of escape mutation within the immunodominant HLA-B*5703-restricted Gag epitope in chronic human immunodeficiency virus type 1 infection. Year Published: 2007
Department of Pediatrics, University of Oxford, Peter Medawar Building for Pathogen Research, South Parks Rd., Oxford OX1 3SY, United Kingdom.
33. Targeting of a CD8 T Cell Env Epitope Presented by HLA-B*5802 Is Associated with Markers of HIV Disease Progression and Lack of Selection Pressure. Year Published: 2008
HIV Pathogenesis Programme, Doris Duke Medical Research Institute, Nelson R. Mandela School of Medicine, University of KwaZulu Natal, Durban 4013, South Africa.
34. Targeting of a CD8 T cell env epitope presented by HLA-B*5802 is associated with markers of HIV disease progression and lack of selection pressure. Year Published: 2008
HIV Pathogenesis Programme, Doris Duke Medical Research Institute, Nelson R. Mandela School of Medicine, University of KwaZulu Natal, Durban 4013, South Africa.
35. Detection of HIV Type 1 Gag-Specific CD4(+) T Cell Responses in Acutely Infected Infants. Year Published: 2008
HIV Pathogenesis Programme, Doris Duke Medical Research Institute, Nelson R. Mandela School of Medicine, University of KwaZulu-Natal, 4013 Durban, South Africa.
36. Detection of HIV type 1 gag-specific CD4(+) T cell responses in acutely infected infants. Year Published: 2008
HIV Pathogenesis Programme, Doris Duke Medical Research Institute, Nelson R. Mandela School of Medicine, University of KwaZulu-Natal, 4013 Durban, South Africa.
37. Lentiviral vectors encoding human immunodeficiency virus type 1 (HIV-1)-specific T-cell receptor genes efficiently convert peripheral blood CD8 T lymphocytes into cytotoxic T lymphocytes with potent in vitro and in vivo HIV-1-specific inhibitory activity. Year Published: 2008
Albert Einstein College of Medicine, Forchheimer Building, Room 408, 1300 Morris Park Ave., Bronx, NY 10461, USA.
38. Evidence of Differential HLA Class I-Mediated Viral Evolution in Functional and Accessory/Regulatory Genes of HIV-1. Year Published: 2008
Partners AIDS Research Center, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, United States of America.
39. Portable flanking sequences modulate CTL epitope processing. Year Published: 2007
Partners AIDS Research Center and Howard Hughes Medical Institute, Massachusetts General Hospital (MGH), Harvard Medical School, Boston, Massachusetts 02129, USA. sylvie_legall@hms.harvard.edu
40. Congenital and acquired long QT syndromes. Year Published: 2003
Division of Cardiology and the Blackburn Genetics Laboratory, Robarts Research Institute, University of Western Ontario, London.
41. Effect of change in posture and exercise on repolarization in patients with long QT syndrome with HERG channel mutations. Year Published: 2005
Division of Cardiology and the Blackburn Genetics Laboratory, Roberts Research Institute, University of Western Ontario, London.