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-Feng-Ting Liu Researcher Activity Profile

Research Author Detailed Information 

profile photo of Feng-Ting LiuFeng-Ting liu researcher

Feng-Ting Liu Publication Rate By Year

Feng-Ting Liu has published 5 paper(s) in 2006, 17 paper(s) in 2007, 2 paper(s) in 2008, for a total of 24 research publications in total.

Feng-Ting Ft Liu Author Information

LAST NAME: liu

FIRST NAME: Feng-Ting

INITIALS: ft

AFFILIATION:

Papers

Feng-Ting Liu's Publication Record

  1. [The modulating effect of proapoptotic protein Bax on the resistance of malignant lymphoma cells to tumor necrosis factor related apoptosis inducing ligand-induced apoptosis] Year Published: 2007
  2. Abdominal Department of Tianjin Cancer Hospital, Tianjin 300060, China.
  3. Molecular basis of interaction between NG2 proteoglycan and galectin-3. Year Published: 2006
  4. Burnham Institute, Developmental Neurobiology Program, La Jolla, California 92037, USA. ywen@burnham.org
  5. [Effects of chemotherapy in recurrent endometrial carcinoma] Year Published: 2006
  6. Center of Gynecologic Oncology, Peking University People's Hospital, Beijing 100044, China.
  7. Cell mechanics using atomic force microscopy-based single-cell compression. Year Published: 2006
  8. Chemistry Department, University of California, Davis, California 95616, USA.
  9. Role of galectin-3 in mast cell functions: galectin-3-deficient mast cells exhibit impaired mediator release and defective JNK expression. Year Published: 2006
  10. Department of Dermatology, School of Medicine, University of California-Davis, Sacramento, CA 95817, USA.
  11. The COOH-terminal globular domain of fibrinogen gamma chain suppresses angiogenesis and tumor growth. Year Published: 2006
  12. Department of Dermatology, University of California-Davis Medical Center, Sacramento, CA 95817, USA.
  13. Galectin-3 stimulates preadipocyte proliferation and is up-regulated in growing adipose tissue. Year Published: 2007
  14. Endocrine Research Unit, Mayo Clinic and Mayo Foundation, Rochester, MN 55905, USA.
  15. An emerging role for galectins in tuning the immune response: lessons from experimental models of inflammatory disease, autoimmunity and cancer. Year Published: 2007
  16. Department of Immunopathology, Institute of Biology and Experimental Medicine (IBYME/ CONICET), Buenos Aires, Argentina. gabyrabi@ciudad.com.ar
  17. Inhibition of Advanced Glycation and Absence of Galectin-3 Prevent Blood-Retinal Barrier Dysfunction during Short-Term Diabetes. Year Published: 2007
  18. Centre for Vision Science, Queen's University Belfast, Royal Victoria Hospital, Grosvenor Road, Belfast BT12 6BA, Northern Ireland, UK.
  19. The role of galectin-3 in promotion of the inflammatory response. Year Published: 2007
  20. Department of Dermatology, University of California, Davis, School of Medicine, Sacramento, California, USA. fliu@ucdavis.edu.
  21. Galectin-3 Functions as an Adhesion Molecule to Support Eosinophil Rolling and Adhesion under Conditions of Flow. Year Published: 2007
  22. Division of Vascular Biology, La Jolla Institute for Molecular Medicine, San Diego, CA 92121.
  23. The role of galectin-3 in promotion of the inflammatory response. Year Published: 2007
  24. Department of Dermatology, University of California, Davis, School of Medicine, Sacramento, California 95816, USA. fliu@ucdavis.edu
  25. Dermal immunopathology: from genetics to effector mechanisms. Year Published: 2007
  26. Department of Medical Microbiology and Immunology, University of Toledo College of Medicine, Toledo, OH, USA, akira.takashima@utoledo.edu.
  27. Mast cells and immunological skin diseases. Year Published: 2007
  28. Department of Dermatology, School of Medicine, University of California, Davis, 3301 C Street, Suite 1400, Sacramento, CA, 95816, USA, fliu@ucdavis.edu.
  29. The role of galectin-3 in promotion of the inflammatory response. Year Published: 2007
  30. Department of Dermatology, University of California, Davis, School of Medicine, Sacramento, California 95816, USA. fliu@ucdavis.edu
  31. Galectin-3 functions as an adhesion molecule to support eosinophil rolling and adhesion under conditions of flow. Year Published: 2007
  32. Division of Vascular Biology, La Jolla Institute for Molecular Medicine, San Diego, CA 92121, USA.
  33. Galectin-3 stimulates preadipocyte proliferation and is up-regulated in growing adipose tissue. Year Published: 2007
  34. Endocrine Research Unit, Mayo Clinic and Mayo Foundation, Rochester, MN 55905, USA.
  35. Inhibition of advanced glycation and absence of galectin-3 prevent blood-retinal barrier dysfunction during short-term diabetes. Year Published: 2007
  36. Centre for Vision Science, Queen's University Belfast, Royal Victoria Hospital, Grosvenor Road, Belfast BT12 6BA, Northern Ireland, UK.
  37. Galectin-3 functions as an adhesion molecule to support eosinophil rolling and adhesion under conditions of flow. Year Published: 2007
  38. Division of Vascular Biology, La Jolla Institute for Molecular Medicine, San Diego, CA 92121, USA.
  39. Dermal immunopathology: from genetics to effector mechanisms. Year Published: 2007
  40. Department of Medical Microbiology and Immunology, University of Toledo College of Medicine, Toledo, OH, USA. akira.takashima@utoledo.edu
  41. Mast cells and immunological skin diseases. Year Published: 2007
  42. Department of Dermatology, School of Medicine, University of California Davis, Sacramento, CA 95816, USA.
  43. Inhibition of advanced glycation and absence of galectin-3 prevent blood-retinal barrier dysfunction during short-term diabetes. Year Published: 2007
  44. Centre for Vision Science, Queen's University Belfast, Royal Victoria Hospital, Grosvenor Road, Belfast BT12 6BA, Northern Ireland, UK.
  45. Increased proteasomal degradation of Bax is a common feature of poor prognosis chronic lymphocytic leukemia. Year Published: 2008
  46. Centre for Haematology, Institute of Cell and Molecular Science, Departments of Haematology and Haemato-Oncology, Queen Mary University of London, UK. s.g.agrawal@qmul.ac.uk
  47. Bortezomib blocks Bax degradation in malignant B cells during treatment with TRAIL. Year Published: 2008
  48. Centre for Haematology, Institute of Cell and Molecular Sciences, Barts and the London, Queen Mary University of London, 4 Newark Street, London, UK.
 

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