-Gregory D Fink Researcher Activity Profile

Research Author Detailed Information 

Gregory D Fink Publication Rate By Year

Gregory D Fink has published 4 paper(s) in 2006, 16 paper(s) in 2007, 2 paper(s) in 2008, for a total of 22 research publications in total.

Gregory D Gd Fink Author Information

LAST NAME: fink

FIRST NAME: Gregory D

INITIALS: gd

AFFILIATION:

Papers

Gregory D Fink's Publication Record

1. The 5-hydroxytryptamine2A receptor is involved in (+)-norfenfluramine-induced arterial contraction and blood pressure increase in deoxycorticosterone acetate-salt hypertension. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48824-1317, USA. niwei@msu.edu
2. Mechanisms of hypertension induced by nitric oxide (NO) deficiency: focus on venous function. Year Published: 2006
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48824-1317, USA.
3. In vitro continuous amperometry with a diamond microelectrode coupled with video microscopy for simultaneously monitoring endogenous norepinephrine and its effect on the contractile response of a rat mesenteric artery. Year Published: 2006
Department of Chemistry, Department of Pharmacology and Toxicology, and the Neuroscience Program, Michigan State University, East Lansing, Michigan 48824, USA.
4. Chronic low-dose angiotensin II infusion increases venomotor tone by neurogenic mechanisms. Year Published: 2006
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48824, USA.
5. Activation of potassium channels by tempol in arterial smooth muscle cells from normotensive and deoxycorticosterone acetate-salt hypertensive rats. Year Published: 2006
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48824, USA. xuhui2@msu.edu
6. A comparison of arteries and veins in oxidative stress: producers, destroyers, function, and disease. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, B445 Life Sciences, East Lansing, MI 48825, USA. szasziri@msu.edu
7. Discordant hemodynamic and fibrinolytic adaptations following a 6-week cardiac rehabilitation program. Year Published: 2007
Department of Kinesiology, Michigan State University, East Lansing, MI, USA. prnagelkirk@bsu.edu
8. Morphological and biochemical characterization of remodeling in aorta and vena cava of DOCA-salt hypertensive rats. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48823-1317, USA. wattss@msu.edu
9. Hypertension caused by prenatal testosterone excess in female sheep. Year Published: 2007
Deparetment of Pharmacology and Toxicology, B440 Life Sciences, Michigan State University, East Lansing, MI 48824, USA. kingand@msu.edu
10. Preferential myosin heavy chain isoform B Expression may contribute to the faster velocity of contraction in veins versus arteries. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48823, USA.
11. Increased sympathetic venoconstriction and reactivity to norepinephrine in mesenteric veins in anesthetized DOCA-salt hypertensive rats. Year Published: 2007
Department of Pharmacology & Toxicology, Neuroscience Program, Michigan State University, East Lansing, MI 48824, USA. xuhui2@msu.edu
12. Splanchnic circulation is a critical neural target in angiotensin II salt hypertension in rats. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48824, USA.
13. Developmental programming of cardiovascular disorders: focus on hypertension. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, E. Lansing, MI 48824, USA. mohankumrs@cvm.msu.edu
14. Discordant hemodynamic and fibrinolytic adaptations following a 6-week cardiac rehabilitation program. Year Published: 2007
Department of Kinesiology, Michigan State University, East Lansing, MI, USA. prnagelkirk@bsu.edu
15. Differences in sympathetic neuroeffector transmission to rat mesenteric arteries and veins as probed by in vitro continuous amperometry and video imaging. Year Published: 2007
Department of Chemistry and the Neuroscience Program, Michigan State University, East Lansing, MI 48824, USA. swain@chemistry.msu.edu.
16. Big ET-1 processing into vasoactive peptides in arteries and veins. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, East Lansing, MI 48824-1317, USA.
17. Discordant hemodynamic and fibrinolytic adaptations following a 6-week cardiac rehabilitation program. Year Published: 2007
Department of Kinesiology, Michigan State University, East Lansing, MI, USA. prnagelkirk@bsu.edu
18. Differences in sympathetic neuroeffector transmission to rat mesenteric arteries and veins as probed by in vitro continuous amperometry and video imaging. Year Published: 2007
Department of Chemistry and the Neuroscience Program, Michigan State University, East Lansing, MI 48824, USA.
19. Developmental programming of cardiovascular disorders: focus on hypertension. Year Published: 2007
Department of Pharmacology and Toxicology, Michigan State University, E. Lansing, MI 48824, USA. mohankumrs@cvm.msu.edu
20. Differences in sympathetic neuroeffector transmission to rat mesenteric arteries and veins as probed by in vitro continuous amperometry and video imaging. Year Published: 2007
Department of Chemistry and the Neuroscience Program, Michigan State University, East Lansing, MI 48824, USA.
21. Loss of the aryl hydrocarbon receptor induces hypoxemia, endothelin-1, and systemic hypertension at modest altitude. Year Published: 2008
College of Pharmacy, MSC09 5360, 2703 Frontier NE, University of New Mexico, Albuquerque, NM 87131. mwalker@salud.unm.edu.
22. Whole body norepinephrine kinetics in ANG II-salt hypertension in the rat. Year Published: 2008
Dept. of Pharmacology and Toxicology, B440 Life Sciences, Michigan State Univ., East Lansing, MI 48824. finkg@msu.edu).