-E Canalis Researcher Activity Profile

Research Author Detailed Information 

E Canalis Publication Rate By Year

E Canalis has published 2 paper(s) in 1985, 1 paper(s) in 1988, 1 paper(s) in 1997, 2 paper(s) in 2000, 1 paper(s) in 2002, 2 paper(s) in 2004, 1 paper(s) in 2005, 3 paper(s) in 2006, 12 paper(s) in 2007, 1 paper(s) in 2008, for a total of 26 research publications in total.

E E Canalis Author Information

LAST NAME: canalis

FIRST NAME: E

INITIALS: e

AFFILIATION:

Papers

E Canalis's Publication Record

1. Transcriptional regulation of connective tissue growth factor by cortisol in osteoblasts. Year Published: 2000
Department of Research, Saint Francis Hospital and Medical Center, Hartford, CT 06105, USA.
2. Bone morphogenetic proteins induce gremlin, a protein that limits their activity in osteoblasts. Year Published: 2000
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105, USA.
3. Effects of cortisol and bone morphogenetic protein-2 on stromal cell differentiation: correlation with CCAAT-enhancer binding protein expression. Year Published: 2002
Department of Research, Saint Francis Hospital and Medical Center, 114 Woodland Street, Hartford, CT 06105-1299, USA.
4. CCAAT/enhancer binding protein homologous protein (DDIT3) induces osteoblastic cell differentiation. Year Published: 2004
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105-1299, USA.
5. Regulation of collagenase-3 by bone morphogenetic protein-2 in bone cell cultures. Year Published: 1997
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105, USA.
6. Bone morphogenetic protein-2 suppresses collagenase-3 promoter activity in osteoblasts through a runt domain factor 2 binding site. Year Published: 2004
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105, USA. svarghes@stfranciscare.org
7. Overexpression of twisted gastrulation inhibits bone morphogenetic protein action and prevents osteoblast cell differentiation in vitro. Year Published: 2005
Department of Research, Saint Francis Hospital and Medical Center, 114 Woodland Street, Hartford, Connecticut 06105-1299, USA.
8. Notch 1 overexpression inhibits osteoblastogenesis by suppressing Wnt/beta-catenin but not bone morphogenetic protein signaling. Year Published: 2006
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105-1299, USA.
9. Expression and regulation of CCN genes in murine osteoblasts. Year Published: 2006
Department of Research, Saint Francis Hospital and Medical Center, 114 Woodland Street, Hartford, CT 06105, USA.
10. Twisted gastrulation, a bone morphogenetic protein agonist/antagonist, is not required for post-natal skeletal function. Year Published: 2006
Department of Research, Saint Francis Hospital and Medical Center, Hartford, CT 06105-1299, USA.
11. CCAAT/Enhancer-binding protein homologous protein (CHOP) decreases bone formation and causes osteopenia. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, CT, USA.
12. Nephroblastoma overexpressed (Nov) inhibits osteoblastogenesis and causes osteopenia. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105, USA.
13. Genetic dissection of mouse distal chromosome 1 reveals three linked BMD QTLs with sex-dependent regulation of bone phenotypes. Year Published: 2007
The Jackson Laboratory, Bar Harbor, Maine, USA. wesley.beamer@jax.org
14. Age-related changes in trabecular architecture differ in female and male C57BL/6J mice. Year Published: 2007
Orthopedic Biomechanics Laboratory, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts 02215, USA.
15. Mechanisms of anabolic therapies for osteoporosis. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, CT 06105-1299, USA. ecanalis@stfranciscare.org
16. Glucocorticoid-induced osteoporosis: pathophysiology and therapy. Year Published: 2007
Saint Francis Hospital and Medical Center, Hartford, CT 060105, USA.
17. Conditional deletion of gremlin causes a transient increase in bone formation and bone mass. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105-1299, USA.
18. Conditional deletion of gremlin causes a transient increase in bone formation and bone mass. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, Connecticut 06105-1299, USA.
19. Mechanisms of anabolic therapies for osteoporosis. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, CT 06105-1299, USA. ecanalis@stfranciscare.org
20. Nephroblastoma overexpressed (nov) is a novel bone morphogenetic protein antagonist. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, 114 Woodland Street, Hartford, CT 06105-1299. ecanalis@stfranciscare.org.
21. Nephroblastoma overexpressed (Nov) is a novel bone morphogenetic protein antagonist. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, CT 06105-1299, USA. ecanalis@stfranciscare.org
22. Effect of growth factors on bone cell replication and differentiation. Year Published: 1985
23. Local bone growth factors. Year Published: 1985
24. Effects of bone associated growth factors on DNA, collagen and osteocalcin synthesis in cultured fetal rat calvariae. Year Published: 1988
Department of Medicine (Endocrine Section), St. Francis Hospital and Medical Center, Hartford 06105.
25. Glucocorticoid-induced osteoporosis: clinical and therapeutic aspects. Year Published: 2008
Department of Internal Medicine, University of Brescia, Italy.
26. Nephroblastoma overexpressed (Nov) is a novel bone morphogenetic protein antagonist. Year Published: 2007
Department of Research, Saint Francis Hospital and Medical Center, Hartford, CT 06105-1299, USA. ecanalis@stfranciscare.org