-David Witte Researcher Activity Profile

Research Author Detailed Information 

David Witte Publication Rate By Year

David Witte has published 1 paper(s) in 2006, 6 paper(s) in 2007, 6 paper(s) in 2008, for a total of 13 research publications in total.

David D Witte Author Information

LAST NAME: witte

FIRST NAME: david

INITIALS: D

AFFILIATION:

Papers

David Witte's Publication Record

1. The proximal convoluted tubule is a target for the uroguanylin-regulated natriuretic response. Year Published: 2006
Division of Gastroenterology, Hepatology and Nutrition, Cincinnati Children's Hospital Medical Center, and the University of Cincinnati, OH 45229, USA.
2. Cdc42 GTPase-activating protein deficiency promotes genomic instability and premature aging-like phenotypes. Year Published: 2007
Division of Experimental Hematology, Molecular Developmental Biology Graduate Program, Children's Hospital Research Foundation, University of Cincinnati, Cincinnati, OH 45229, USA.
3. Effect of rotavirus strain on the murine model of biliary atresia. Year Published: 2007
Cincinnati Children's Hospital Medical Center, 3333 Burnet Avenue, MLC 2923, Cincinnati, OH 45229, USA.
4. Identification of S664 TSC2 phosphorylation as a marker for extracellular signal-regulated kinase mediated mTOR activation in tuberous sclerosis and human cancer. Year Published: 2007
Cancer Biology and Genetics Program, Sloan-Kettering Institute, Memorial Sloan-Kettering Cancer Center, New York, NY 10021, USA.
5. Fibrinogen deficiency, but not plasminogen deficiency, increases mortality synergistically in combination with sickle hemoglobin SAD in transgenic mice. Year Published: 2007
Comprehensive Sickle Cell Center, Children's Hospital Medical Center, Cincinnati, Ohio.
6. Fibrinogen deficiency, but not plasminogen deficiency, increases mortality synergistically in combination with sickle hemoglobin SAD in transgenic mice. Year Published: 2007
Comprehensive Sickle Cell Center, Children's Hospital Medical Center, Cincinnati, Ohio 45229, USA.
7. Fibrinogen deficiency, but not plasminogen deficiency, increases mortality synergistically in combination with sickle hemoglobin SAD in transgenic mice. Year Published: 2007
Comprehensive Sickle Cell Center, Children's Hospital Medical Center, Cincinnati, Ohio 45229, USA.
8. Plexiform and dermal neurofibromas and pigmentation are caused by nf1 loss in desert hedgehog-expressing cells. Year Published: 2008
Division of Experimental Hematology and Cancer Biology, Cincinnati Children's Research Foundation, Cincinnati Children's Hospital Medical Center, University of Cincinnati College of Medicine, Cincinnati, OH 45229, USA.
9. Rac1 is essential for intraembryonic hematopoiesis and for the initial seeding of fetal liver with definitive hematopoietic progenitor cells. Year Published: 2008
10. Plexiform and dermal neurofibromas and pigmentation are caused by Nf1 loss in desert hedgehog-expressing cells. Year Published: 2008
Division of Experimental Hematology and Cancer Biology, Cincinnati Children's Research Foundation, Cincinnati Children's Hospital Medical Center, University of Cincinnati College of Medicine, Cincinnati, OH 45229, USA.
11. Novel fibrillin 1 mutation in a case of neonatal marfan syndrome: the increasing importance of early recognition. Year Published: 2008
Division of Cardiology, Cincinnati Children's Hospital Medical Center, Cincinnati, Ohio, USA.
12. Novel fibrillin 1 mutation in a case of neonatal Marfan syndrome: the increasing importance of early recognition. Year Published: 2008
Division of Cardiology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229-3039, USA.
13. Rac1 is essential for intraembryonic hematopoiesis and for the initial seeding of fetal liver with definitive hematopoietic progenitor cells. Year Published: 2008
Department of Pediatrics, Cincinnati Children's Research Foundation and Cincinnati Children's Hospital Medical Center, and Hoxworth Blood Center, University of Cincinnati College of Medicine, OH, USA.