-Hartmut Jaeschke Researcher Activity Profile

Research Author Detailed Information 

Hartmut Jaeschke Publication Rate By Year

Hartmut Jaeschke has published 3 paper(s) in 2006, 9 paper(s) in 2007, 1 paper(s) in 2008, for a total of 13 research publications in total.

Hartmut H Jaeschke Author Information

LAST NAME: jaeschke

FIRST NAME: hartmut

INITIALS: H

AFFILIATION:

Papers

Hartmut Jaeschke's Publication Record

1. Role of neutrophils in acute inflammatory liver injury. Year Published: 2006
Liver Research Institute, University of Arizona, College of Medicine, Tucson, AZ 85737, USA. jaeschke@email.arizona.edu
2. Pathophysiological role of the acute inflammatory response during acetaminophen hepatotoxicity. Year Published: 2006
Liver Research Institute, University of Arizona, Tucson, AZ 85724, USA.
3. Nuclear translocation of endonuclease G and apoptosis-inducing factor during acetaminophen-induced liver cell injury. Year Published: 2006
Liver Research Institute, University of Arizona, College of Medicine, Tucson, Arizona 85724, USA.
4. Reduced inflammatory response and increased microcirculatory disturbances during hepatic ischemia-reperfusion injury in steatotic livers of ob/ob mice. Year Published: 2007
Liver Research Institute, College of Medicine, University of Arizona, Tucson, Arizona, USA.
5. Hepatobiliary transporter expression in intercellular adhesion molecule 1 knockout and Fas receptor-deficient mice after common bile duct ligation is independent of the degree of inflammation and oxidative stress. Year Published: 2007
Laboratory of Experimental and Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Medicine, Medical University Graz, Auenbruggerplatz 15, A-8036 Graz, Austria.
6. Role of neutrophils in the pathogenesis of acute inflammatory liver injury. Year Published: 2007
Department of Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University, College Station, TX, USA.
7. Hepatobiliary transporter expression in intercellular adhesion molecule 1 knockout and Fas receptor-deficient mice after common bile duct ligation is independent of the degree of inflammation and oxidative stress. Year Published: 2007
Laboratory of Experimental and Molecular Hepatology, Division of Gastroenterology and Hepatology, Department of Medicine, Medical University Graz, Auenbruggerplatz 15, A-8036 Graz, Austria.
8. Role of neutrophils in the pathogenesis of acute inflammatory liver injury. Year Published: 2007
Department of Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University, College Station, TX 77843-4467, USA. sramaiah@cvm.tamu.edu
9. Mitochondrial bax translocation accelerates DNA fragmentation and cell necrosis in a murine model of acetaminophen hepatotoxicity. Year Published: 2007
Department of Pharmacology, Toxicology, and Therapeutics, University of Kansas Medical Center, 3901 Rainbow Blvd., MS 1018, Kansas City, KS 66160, USA.
10. Mitochondrial bax translocation accelerates DNA fragmentation and cell necrosis in a murine model of acetaminophen hepatotoxicity. Year Published: 2007
Department of Pharmacology, Toxicology, and Therapeutics, University of Kansas Medical Center, 3901 Rainbow Blvd., MS 1018, Kansas City, KS 66160, USA.
11. Role of neutrophils in the pathogenesis of acute inflammatory liver injury. Year Published: 2007
Department of Pathobiology, College of Veterinary Medicine & Biomedical Sciences, Texas A&M University, College Station, TX 77843-4467, USA. sramaiah@cvm.tamu.edu
12. Mitochondrial bax translocation accelerates DNA fragmentation and cell necrosis in a murine model of acetaminophen hepatotoxicity. Year Published: 2007
Department of Pharmacology, Toxicology, and Therapeutics, University of Kansas Medical Center, 3901 Rainbow Blvd., MS 1018, Kansas City, KS 66160, USA.
13. Mitochondrial protein thiol modifications in acetaminophen hepatotoxicity: Effect on HMG-CoA synthase. Year Published: 2008
Department of Environmental Health Sciences and The Center for Free Radical Biology, University of Alabama at Birmingham, Birmingham, AL 35294, United States.