-Costantino Iadecola Researcher Activity Profile

Research Author Detailed Information 

Costantino Iadecola Publication Rate By Year

Costantino Iadecola has published 18 paper(s) in 2007, 6 paper(s) in 2008, for a total of 24 research publications in total.

Costantino C Iadecola Author Information

LAST NAME: iadecola

FIRST NAME: costantino

INITIALS: C

AFFILIATION:

Papers

Costantino Iadecola's Publication Record

1. Cerebrovascular nitrosative stress mediates neurovascular and endothelial dysfunction induced by angiotensin II. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, New York, NY 10021, USA.
2. Testosterone production in mice lacking inducible nitric oxide synthase expression is sensitive to restraint stress. Year Published: 2007
Department of Pharmacology, Israel Institute for Biological Research, Ness Ziona, Israel. baw@iibr.gov.il
3. Cyclooxygenase-2 does not contribute to postischemic production of reactive oxygen species. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, New York, NY 10021, USA.
4. Cellular and subcellular localization of androgen receptor immunoreactivity relative to C1 adrenergic neurons in the rostral ventrolateral medulla of male and female rats. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, New York 10021, USA. tmilner@mail.med.cornell.edu
5. Changes in the subcellular distribution of NADPH oxidase subunit p47phox in dendrites of rat dorsomedial nucleus tractus solitarius neurons in response to chronic administration of hypertensive agents. Year Published: 2007
Department of Neurology and Neuroscience, Weill Medical College of Cornell University, 411 E. 69th St., KB410, New York, NY 10021, USA. mjg2003@mail.med.cornell.edu
6. Neurovascular protection by ischemic tolerance: role of nitric oxide and reactive oxygen species. Year Published: 2007
Division of Neurobiology, Weill-Cornell Medical College, KB-410, New York, New York 10021, USA.
7. iNOS-derived NO and nox2-derived superoxide confer tolerance to excitotoxic brain injury through peroxynitrite. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, New York, New York 10021, USA.
8. MyD88-5 links mitochondria, microtubules, and JNK3 in neurons and regulates neuronal survival. Year Published: 2007
Department of Microbiology and Immunology, Weill Medical College of Cornell University, New York, NY 10021, USA.
9. Lipopolysaccharide induces early tolerance to excitotoxicity via nitric oxide and cGMP. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, 411 East 69th St, KB-410, New York, NY 10021, USA.
10. Heat shock proteins and p53 play a critical role in K+ channel-mediated tumor cell proliferation and apoptosis. Year Published: 2007
Cancer Biology Reseach Center, Tongji Hospital, Huazhong University of Science and Technology, Wuhan, Hubei, 430030, China.
11. Glial regulation of the cerebral microvasculature. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Cornell Medical College, 411 East 69th St., New York, New York 10021, USA.
12. Nox2-derived reactive oxygen species mediate neurovascular dysregulation in the aging mouse brain. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, New York 10021, USA.
13. Farnesyl transferase inhibitors induce neuroprotection by inhibiting Ha-Ras signalling pathway. Year Published: 2007
Department of Clinical and Experimental Medicine, Federico II University, Naples, Italy, abd Emergency Cardiology, Cardarelli Hospital, Naples, Italy.
14. Glial regulation of the cerebral microvasculature. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Cornell Medical College, New York, New York 10021, USA. coi2001@med.cornell.edu
15. Nox2-derived reactive oxygen species mediate neurovascular dysregulation in the aging mouse brain. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, New York 10021, USA.
16. Farnesyl transferase inhibitors induce neuroprotection by inhibiting Ha-Ras signalling pathway. Year Published: 2007
Department of Clinical and Experimental Medicine, Federico II University, Naples, Italy.
17. Nox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10065, USA.
18. Key role of tissue plasminogen activator in neurovascular coupling. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10021.
19. Key role of tissue plasminogen activator in neurovascular coupling. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10021, USA.
20. Glial regulation of the cerebral microvasculature. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Cornell Medical College, New York, New York 10021, USA. coi2001@med.cornell.edu
21. Key role of tissue plasminogen activator in neurovascular coupling. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10021, USA.
22. Nuclear Factor-{kappa}B Activation and Postischemic Inflammation Are Suppressed in CD36-Null Mice after Middle Cerebral Artery Occlusion. Year Published: 2008
Division of Neurobiology, Weill Cornell Medical College, New York, New York 10021.
23. Nuclear factor-kappaB activation and postischemic inflammation are suppressed in CD36-null mice after middle cerebral artery occlusion. Year Published: 2008
Division of Neurobiology, Weill Cornell Medical College, New York, New York 10021, USA.
24. Heat shock proteins and p53 play a critical role in K+ channel-mediated tumor cell proliferation and apoptosis. Year Published: 2007
Cancer Biology Reseach Center, Tongji Hospital, Huazhong University of Science and Technology, Wuhan, Hubei, 430030, China.