-Josef Anrather Researcher Activity Profile

Research Author Detailed Information 

Josef Anrather Publication Rate By Year

Josef Anrather has published 15 paper(s) in 2007, 6 paper(s) in 2008, for a total of 21 research publications in total.

Josef J Anrather Author Information

LAST NAME: anrather

FIRST NAME: josef

INITIALS: J

AFFILIATION:

Papers

Josef Anrather's Publication Record

1. Cerebrovascular nitrosative stress mediates neurovascular and endothelial dysfunction induced by angiotensin II. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, New York, NY 10021, USA.
2. Cyclooxygenase-2 does not contribute to postischemic production of reactive oxygen species. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, New York, NY 10021, USA.
3. Neurovascular protection by ischemic tolerance: role of nitric oxide and reactive oxygen species. Year Published: 2007
Division of Neurobiology, Weill-Cornell Medical College, KB-410, New York, New York 10021, USA.
4. iNOS-derived NO and nox2-derived superoxide confer tolerance to excitotoxic brain injury through peroxynitrite. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, New York, New York 10021, USA.
5. Lipopolysaccharide induces early tolerance to excitotoxicity via nitric oxide and cGMP. Year Published: 2007
Division of Neurobiology, Weill Medical College of Cornell University, 411 East 69th St, KB-410, New York, NY 10021, USA.
6. Nox2-derived reactive oxygen species mediate neurovascular dysregulation in the aging mouse brain. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, New York 10021, USA.
7. Hypo-phosphorylation leads to nuclear retention of NF-kappaB p65 due to impaired IkappaBalpha gene synthesis. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, 411 East 69th Street, KB410, New York NY10021, USA.
8. Heme Oxygenase-1 Inhibits the Expression of Adhesion Molecules Associated with Endothelial Cell Activation via Inhibition of NF-{kappa}B RelA Phosphorylation at Serine 276. Year Published: 2007
9. Farnesyl transferase inhibitors induce neuroprotection by inhibiting Ha-Ras signalling pathway. Year Published: 2007
Department of Clinical and Experimental Medicine, Federico II University, Naples, Italy, abd Emergency Cardiology, Cardarelli Hospital, Naples, Italy.
10. Nox2-derived reactive oxygen species mediate neurovascular dysregulation in the aging mouse brain. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, New York 10021, USA.
11. Heme oxygenase-1 inhibits the expression of adhesion molecules associated with endothelial cell activation via inhibition of NF-kappaB RelA phosphorylation at serine 276. Year Published: 2007
12. Farnesyl transferase inhibitors induce neuroprotection by inhibiting Ha-Ras signalling pathway. Year Published: 2007
Department of Clinical and Experimental Medicine, Federico II University, Naples, Italy.
13. Hypo-phosphorylation leads to nuclear retention of NF-kappaB p65 due to impaired IkappaBalpha gene synthesis. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, 411 East 69th Street, New York, NY 10021, USA.
14. Nox2-derived radicals contribute to neurovascular and behavioral dysfunction in mice overexpressing the amyloid precursor protein. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10065, USA.
15. Key role of tissue plasminogen activator in neurovascular coupling. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10021.
16. Key role of tissue plasminogen activator in neurovascular coupling. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10021, USA.
17. Hypo-phosphorylation leads to nuclear retention of NF-kappaB p65 due to impaired IkappaBalpha gene synthesis. Year Published: 2007
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, 411 East 69th Street, New York, NY 10021, USA.
18. Heme oxygenase-1 inhibits the expression of adhesion molecules associated with endothelial cell activation via inhibition of NF-kappaB RelA phosphorylation at serine 276. Year Published: 2007
Instituto Gulbenkian de Ciência, Apartado 14, Oeiras, Portugal.
19. Key role of tissue plasminogen activator in neurovascular coupling. Year Published: 2008
Division of Neurobiology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10021, USA.
20. Nuclear Factor-{kappa}B Activation and Postischemic Inflammation Are Suppressed in CD36-Null Mice after Middle Cerebral Artery Occlusion. Year Published: 2008
Division of Neurobiology, Weill Cornell Medical College, New York, New York 10021.
21. Nuclear factor-kappaB activation and postischemic inflammation are suppressed in CD36-null mice after middle cerebral artery occlusion. Year Published: 2008
Division of Neurobiology, Weill Cornell Medical College, New York, New York 10021, USA.