Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features.

Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features. Abstract Text:

Akiko Sugiyama,Mitsuhiro Sugiura,Adriano Piris,Yasushi Tomita,Martin C Mihm,Akiko Sugiyama,Mitsuhiro Sugiura,Adriano Piris,Yasushi Tomita,Martin C Mihm,Akiko Sugiyama,Mitsuhiro Sugiura,Adriano Piris,Yasushi Tomita,Martin C Mihm,

Background: Apocrine cystadenoma (AC) and apocrine hidrocystoma (AH) have been used interchangeably in the literature to designate cystic lesions of apocrine glands. Methods: We reviewed 21 cases with biopsies of apocrine cystic lesions diagnosed as AH or AC stained by hematoxylin and eosin. The following histological characteristics were recorded: (a) number of cysts, (b) predominant architectural growth pattern of cyst wall, (c) tumor circumscription, (d) nuclear atypia, (e) mitotic activity, counted per 1 mm(2) and (f) Ki-67 staining pattern. Results: Our findings clearly show that there is a non-proliferative group and a proliferative group among the lesions. In the non-proliferative group, one may see some structures that resemble papillary projections but lack a fibrous core. In the proliferative group, we found true papillae, and this change was associated with atypia, mitotic activity and increased Ki-67 staining. Conclusions: Apocrine cystic lesions with true papillary projections should be referred to as AC rather than AH, to emphasize the proliferative adenomatous growth and depicted by their frequency of cytological atypia and high mitotic activity. Furthermore, we suggest complete excision of AC that are proliferative tumors.

Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features. Publishing Authors By Initials

A Sugiyama,M Sugiura,A Piris,Y Tomita,MC Mihm,A Sugiyama,M Sugiura,A Piris,Y Tomita,MC Mihm,A Sugiyama,M Sugiura,A Piris,Y Tomita,MC Mihm,

For similar abstracts research abstracts see: abstracts research

PUBMED ID PMID:

MEDLINE DATE:

Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features. Journal Published:

PUBLICATION TYPE: Journal Article

Journal: Journal of cutaneous pathology

VOLUME: 34

Page Numbers: 912-7

Journal Abbreviation: J. Cutan. Pathol.

ISSN: 0303-6987

DAY: 16

MONTH: Dec

YEAR: 2007

Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features. Information

Number of References:

LANGUAGE: eng

NlmUniqueID: 425124

Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features. Keywords Mesh Terms:

KEYWORDS:

MESH TERMS:

Chemical & Substance for Abstract: Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features. Information

Substance Name:

Registry Number:

Grant and Affiliation Information for Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features.

AFFILIATION: Department of Dermatology, Nagoya University Graduate School of Medicine, Nagoya, Japan.

Country: Denmark

AGENCY:

GRANT:

ACRONYM:

MEDLINETA: J Cutan Pathol

REFSOURCE:

DATABASENAME:

ACCESSION NUMBER:

Number Hits: 0

Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features Related Publications

• A unique microvascular phenotype shared by juvenile hemangiomas and human placenta.
• Apocrine cystadenoma and apocrine hidrocystoma: examination of 21 cases with emphasis on nomenclature according to proliferative features.
• CTLA-4 blockade with ipilimumab induces significant clinical benefit in a female with melanoma metastases to the CNS.
• Congenital nonprogressive hemangioma: a distinct clinicopathologic entity unlike infantile hemangioma.
• Frequency of dysplastic nevi among nevomelanocytic lesions submitted for histopathologic examination. Time trends over a 37-year period.
• Infantile hemangioma: clinical resolution with 5% imiquimod cream.
• Interface dermatitis.
• Investigation of the vitamins A and E and beta-carotene content in milk from UK organic and conventional dairy farms.
• Linear IgA bullous dermatosis v dermatitis herpetiformis. Quantitative measurements of dermoepidermal alterations.
• Melanocytic proliferations associated with lichen sclerosus.
• Microvascular injury in lymphomatoid granulomatosis involving the skin. An ultrastructural study.
• Multiple melanocytoma.
• Neutrophilic eccrine hidradenitis. A distinctive type of neutrophilic dermatosis associated with myelogenous leukemia and chemotherapy.
• Oral warty dyskeratoma.
• Origin of cutaneous melanoma in a congenital dysplastic nevus spilus.
• The molecular basis of melanomagenesis and the metastatic phenotype.
• The nonrandom distribution of facial hemangiomas.
• The precursors of malignant melanoma.
• Unusual histologic and clinical variants of melanoma: implications for therapy.
• Vulvar nevi, lichen sclerosus et atrophicus, and vitiligo.