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Anterior canal benign paroxysmal positional vertigo: an underappreciated entity.

Anterior canal benign paroxysmal positional vertigo: an underappreciated entity. Research Abstract Details 

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  • Anterior canal benign paroxysmal positional vertigo: an underappreciated entity. Abstract Text:

    douglas g hetzlerDouglas G Hetzler,

    OBJECTIVE: Evaluate the frequency and characteristics of benign paroxysmal positional vertigo (BPPV) arising from involvement of the anterior semicircular canal (AC) as compared with the posterior canal (PC) and horizontal canal (HC). STUDY DESIGN: Prospective review of patients with BPPV. SETTING: Tertiary referral center. PATIENTS: A total of 260 patients who were evaluated for vertigo were identified as experiencing BPPV. INTERVENTIONS: Standard vestibular assessment including the use of electrooculography (EOG) or video-oculography (VOG) was completed on all patients. Based on EOG/VOG findings, the BPPV origin was attributed to AC, PC, or HC involvement secondary to canalithiasis versus cupulolithiasis. Treatment was performed with canalith repositioning maneuvers (CRMs) appropriate for type of canal involvement. RESULTS: For the 260 patients, the positionally induced nystagmus patterns suggested the canal of origin to be AC in 21.2%, PC in 66.9%, and HC in 11.9%. Cupulolithiasis was observed in 27.3% of the AC, 6.3% of the PC, and 41.9% of the HC patients. Head trauma was confirmed in the history preceding the onset of vertigo in 36.4% of the AC, versus 9.2% of the PC and 9.7% of the HC patients (p < 0.001). The number of CRMs completed to treat the BPPV did not differ between canals involved (1.32 for AC, 1.49 for PC, and 1.34 for HC). CONCLUSION: The direction of subtle vertical-beating nystagmus underlying the torsional component is critical in differentiating AC versus PC origin; EOG/VOG aids in accurate assessment of the vertical component for the diagnosis of canal involvement. AC involvement may be more prevalent than previously appreciated, particularly if the examiner does not appreciate the vertical component of the nystagmus or the diagnosis is made without the assistance of EOG/VOG. Head trauma history is significantly more frequent in AC versus other forms of BPPV, and patients with a history of head trauma should be examined closely for AC involvement. CRM is as successful for treatment of AC BPPV as for other types of BPPV.

    Anterior canal benign paroxysmal positional vertigo: an underappreciated entity. Publishing Authors By Initials

    dg hetzlerDG Hetzler,

    For similar technology, industry, and agriculture: technology: educational technology: audiovisual aids: tape recording: videotape recording research abstracts see: technology, industry, and agriculture: technology: educational technology: audiovisual aids: tape recording: videotape recording research

    PUBMED ID PMID:

    MEDLINE DATE:

    Anterior canal benign paroxysmal positional vertigo: an underappreciated entity. Journal Published:

    PUBLICATION TYPE: Journal Article

    Journal: Otology & neurotology : official publication of th

    VOLUME: 28

    Page Numbers: 218-22

    Journal Abbreviation: Otol. Neurotol.

    ISSN: 1531-7129

    DAY: 20

    MONTH: Feb

    YEAR: 2007

    Anterior canal benign paroxysmal positional vertigo: an underappreciated entity. Information

    Number of References:

    LANGUAGE: eng

    NlmUniqueID: 100961504

    Anterior canal benign paroxysmal positional vertigo: an underappreciated entity. Keywords Mesh Terms:

    KEYWORDS: Videotape Recording

    MESH TERMS: physiopathology

    Chemical & Substance for Abstract: Anterior canal benign paroxysmal positional vertigo: an underappreciated entity. Information

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    Grant and Affiliation Information for Anterior canal benign paroxysmal positional vertigo: an underappreciated entity.

    AFFILIATION: Ear Institute of Texas, San Antonio, TX 78240, USA. Ljacksonmd@EIofTX.com

    Country: United States

    United States Research PublicationUnited States Research Publication

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    MEDLINETA: Otol Neurotol

    REFSOURCE: Otol Neurotol. 2007 Oct;28(7):995; autho

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