Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits.

Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits. Research Abstract Details 

Research Abstract Table of Contents

Jump to the:

Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits. Abstract Text:

Wei Zhong,Linda Levin,Tiina Reponen,Gurjit K Hershey,Atin Adhikari,Rakesh Shukla,Grace LeMasters,Wei Zhong,Linda Levin,Tiina Reponen,Gurjit K Hershey,Atin Adhikari,Rakesh Shukla,Grace LeMasters,

The objective of our study was to investigate the association between daily pediatric asthma hospital visits and daily concentrations of aeroallergens and their specific species. Records of daily asthma visits in Cincinnati area were retrieved from Cincinnati Children's Hospital Medical Center and aeroallergen sampling was performed by the Button Inhalable Sampler. The Poisson generalized linear model was carried out in which the log of the number of asthma hospital visits was related to aeroallergen level, treated as a continuous variable with adjustment for seasonal time trend, day of the week, ozone and PM(2.5) concentrations, temperature and humidity. The aeroallergens having a significant impact on asthma hospital visits were ragweed, oak/maple and Pinaceae pollen. Their relative risks on asthma hospital visits with respect to a 100 counts/m(3) increase in concentration were in the range of 1.23 to 1.54. The effects in causing the asthma exacerbation were delayed by 3 or 5 days.

Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits. Publishing Authors By Initials

W Zhong,L Levin,T Reponen,GK Hershey,A Adhikari,R Shukla,G LeMasters,W Zhong,L Levin,T Reponen,GK Hershey,A Adhikari,R Shukla,G LeMasters,

For similar cells: spores: spores, fungal research abstracts see: cells: spores: spores, fungal research

PUBMED ID PMID:

MEDLINE DATE:

Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits. Journal Published:

PUBLICATION TYPE: Research Support, N.I.H., Extr

Journal: The Science of the total environment

VOLUME: 370

Page Numbers: 330-6

Journal Abbreviation: Sci. Total Environ.

ISSN: 0048-9697

DAY: 14

MONTH: 08

YEAR: 2006

Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits. Information

Number of References:

LANGUAGE: eng

NlmUniqueID: 330500

Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits. Keywords Mesh Terms:

KEYWORDS: Spores, Fungal

MESH TERMS: isolation & purification

Chemical & Substance for Abstract: Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits. Information

Substance Name: Ozone

Registry Number: 10028-15-6

Grant and Affiliation Information for Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits.

AFFILIATION: Department of Environmental Health, University of Cincinnati, PO Box 670056, Ohio 45267-0056, USA. weizhong22@yahoo.com

Country: Netherlands

AGENCY: United States NIEHS

GRANT: R01 ES 1170

ACRONYM: ES

MEDLINETA: Sci Total Environ

REFSOURCE:

DATABASENAME:

ACCESSION NUMBER:

Number Hits: 0

Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits Related Publications

• A review of land-use regression models for characterizing intraurban air pollution exposure.
• Activation of the oxygen-sensing signal cascade prevents mitochondrial injury after mouse liver ischemia-reperfusion.
• Analysis of short-term influences of ambient aeroallergens on pediatric asthma hospital visits.
• C-Jun N-Terminal Kinase 2 Promotes Graft Injury via the Mitochondrial Permeability Transition After Mouse Liver Transplantation.
• Different patterns of renal cell killing after warm and cold ischemia.
• Endothelial nitric oxide synthase protects transplanted mouse livers against storage/reperfusion injury: Role of vasodilatory and innate immunity pathways.
• Estimating sampling frequency in pollen exposure assessment over time.
• Graft tumor necrosis factor receptor-1 protects after mouse liver transplantation whereas host tumor necrosis factor receptor-1 promotes injury.
• House dust (1-3)-beta-D-glucan and wheezing in infants.
• Kupffer cell-dependent reperfusion injury in liver transplantation: new clinically relevant use of glycine.
• Minocycline and N-methyl-4-isoleucine cyclosporin (NIM811) mitigate storage/reperfusion injury after rat liver transplantation through suppression of the mitochondrial permeability transition.
• Mitochondrial Permeability Transition in Liver Ischemia and Reperfusion: Role of c-Jun N-Terminal Kinase 2.
• Modulation of mitochondrial membrane permeability in pathogenesis, autophagy and control of metabolism.
• NIM811, a mitochondrial permeability transition inhibitor, prevents mitochondrial depolarization in small-for-size rat liver grafts.
• Selective degradation of mitochondria by mitophagy.
• The effect of home characteristics on dust antigen concentrations and loads in homes.
• c-Jun N-terminal kinase mediates hepatic injury after rat liver transplantation.